Pineal Gland Activation Course

Pineal Gland Activation Course

World renowned expert on the pineal gland, Shaktipat Seer has helped thousands around the planet activate their pineal gland safely. Through pineal gland activation you can have a thorough cleansing of your aura, bringing into full effect the latent kundalini powers of your body. Over the years he has perfected his ability to give direct transmission of Spiritual Energy to the Third Eye Chakra, setting alight the glowing powers of the Philosophers Stone of the Neo Cortex region. Discover A Simple System That Anyone Can Do, Regardless Of Age Or Ability And From The Comfort Of Their Own Home. Through Pineal Gland Activation You Can Have A Cleansing Of Your Aura, Bringing Into Full Effect The Latent Kundalini Powers Of The Body Leading To. Shaktipat Seer is not trying to give you some cooky pseudo-science that many snake oil salesman push that has no real transcendental benefit. Instead he is merely presenting the natural way that this process has been effected (through transmission of Shakti to the Third Eye) in the East (India,Tibet,China etc.) for thousands and thousands of years.

Pineal Gland Activation Course Overview


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Contents: Ebook, 14 Day Course
Author: Shaktipat Seer
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The Pineal Gland and Melatonin

The pineal gland is a neuroendocrine gland that synthesizes and secretes melatonin ( N-acetyl-5-methoxytryptamine). y The afferent input to the pineal gland is transmitted from the retinal photoreceptors through the SCN and sympathetic nervous system. The circadian rhythm of melatonin is controlled by the SCN but is strongly entrained by light. The two effects of light are, first, to regulate melatonin secretion in accordance with diurnal light-dark cycles and, second, to suppress melatonin if given in brief intense pulses. Melatonin secretion increases during the night, reaching a peak level between 2 00 and 4 00 am, then gradually falls during the latter part of the night, and is present at very low levels during the day. Exogenous melatonin has been used with some success to avoid jet lag and may be useful for treatment of phase-shifted sleep and sleep disturbance due to shift work. Melatonin is available through health food stores and has received strong public attention. However,...

Input to Pineal Gland and Sites of Action of Melatonin

The foremost neuronal input to PG consists of a multisynaptic pathway named the retinohypotalamic-pineal tract. The first step of this pathway is represented by the previously mentioned RHT, which comes from the retina and ends in the ventrolateral SCN (55,56). The endogenous pacemaker (SCN) projects inhibitory and excitatory neurons to the PVN mediated by GABA and VIP VP, respectively (57). The PVN projects to the intermediolateral neurons of the first three cervical spinal levels via oxytocin and VP, where the injection of these two neurotransmitters to this spinal area decreases MLT plasma concentrations (58). From there, cholinergic excitatory neurons reach the superior cervical ganglion that projects sympathetic innervation to the PG (59). Besides norepinephrine, these nerve endings contain dopamine, serotonin, VIP, and neuropeptide Y (NPY). Other neuronal input to the PG originated from the thalamic IGL via NPY, the PVN via VP and oxytocin, the lateral hypothalamus via...

Melatonin as a Circadian Hormone

FIGURE 1 Schematic organization of nervous network for circadian control. Light exerts its entrainment on SCN by the RHT. The nervous terminal of this pathway contains GLU and SP. Thanks to its autodepolarization property, the SCN represents the endogenous pacemaker. VIP, VP, and GABA are the main neurotrasmitters for the SCN efferents to VMN, SPZ, and PVN. SCN is also connected by a reciprocal innervation to IGL. SNC acts on PG through the multisynaptic retinohypothalamic-pineal pathway, which is represented in the figure by sketched line. Other inputs to PG are from IGL, LH, and HN. PG produces MLT, which reaches its targets via paracrine and endocrine secretion. MLT receptors are located in several neuronal and extraneuronal tissues. Abbreviations RT, retina GHT, geniculohypothalamic tract IGL, intergeniculate leaflet RHT, retino-hypothalamic tract SCN, suprachiasmatic nucleus VMN, ventromedialis nucleus SPZ, subparaventricular zone PVN, paraventricular nucleus CSC, cervical spinal...

Clinical features

There is a tendency towards an advanced sleep phase syndrome in depression (Fig. 7.3) which may contribute to early morning awakening 12 . In contrast, depression in the seasonal affective disorder causes excessive daytime sleepiness and a delayed sleep phase syndrome. Changes in circadian rhythms such as cortisol secretion are common in depression. Melatonin secretion is reduced in depression, possibly because of reduced 5HT and noradrenaline stimulation of the pineal gland. The amplitude of the diurnal temperature rhythm is lessened and core body temperature at night is higher than in normal subjects. The reduction in REM sleep latency correlates with both of these abnormalities.

Normal Organ Development

The axial skeleton consists of the skull, the vertebrae, the sternum, and the ribs. The bones of the limbs make up the appendicular skeleton. The bones of the axial skeleton are flat or irregularly shaped. Most of the bones of the appendicular skeleton are long bones (see Fig. 16.1) 64 and have a shaft (dia-physis), a medullary cavity and two enlarged ends (epiphyses). The epiphysis at each end extends from articular cartilage to the epiphyseal growth plate. The metaphysis is the region between the epiphyseal plate and the diaphysis. After initial ossification in utero, longitudinal growth of the bone occurs only at the epiphyseal plate (physis). The mechanism of growth at the physis is the proliferation of a layer of chon-droblasts, which in turn form a layer of cartilage (Fig. 16.2). Small blood vessels invade the cartilage, increasing oxygen tension and stimulating the formation of osteoblasts. The osteoblasts create osteoid that calcifies into bone 60,61 . Schematic of a growing...

Organ Damage Induced by Cytotoxic Therapy

A close-up of the region of the meta-physis and epiphysis.The proliferating cells (chondroblasts) are shown in the region of the physis. (From 61 .) There are too many variables to determine the effect of each individually. In general, though, if the radiation dose is fractionated normally (1-2 Gy per day), the total dose is one of the most important factors. The epiphyseal plate is the most sensitive structure, although for total doses of less than 10 Gy, there are few detectable long-term changes. Doses of 10-20 Gy will produce partial growth arrest of the epiphysis. Doses of greater than 20 Gy will usually result in complete arrest. However, the response to radiation is not an all-or-nothing phenomena the higher the total dose and the younger the age at treatment, the greater the ultimate deficit 65 . This is

Management of Established Problems 16251 Management

Table 16.2 shows the recommended treatment for categories of leg-length discrepancies 39,83 . Small differences (0-2 cm) usually require no intervention. Greater differences require an orthopedic evaluation. Differences of 2-6 cm can be corrected with a shoe lift or a contralateral epiphysiodesis, an operation creating a premature fusion of an epiphysis in the contralateral limb to arrest growth. This prevents further exaggeration of the deficit. Greater inequality (6-15 cm) requires more aggressive management. Contralateral limb shortening or ipsilateral lengthening procedures are usually necessary to restore a functional gait. Differences of greater than 15-20 cm are difficult to manage. The occurrence of a slipped capitofemoral epiph-ysis is a medical emergency requiring immediate referral to an orthopedic surgeon. Correcting the problem requires an in situ pin fixation to prevent a slipped capitofemoral epiphysis in the other leg if it also has been irradiated. Prophylactic...

Basis for Sports Nutrition Interest 1 Insulinomimetic Actions

One of the first vanadium-deprivation signs reported for chicks was adverse effects on bone devel-opment.206 Histological examination of the tibias from vanadium-deprived chicks revealed severe disorganization of the cells of the epiphysis. The cells appeared compressed and their nuclei flattened. These abnormalities apparently were the reason that vanadium-deprived chicks had a shortened, thickened leg structure. Bone abnormalities were also found in vanadium-deprived goats.205 Compared with goats fed a 0.5-2-mg V kg diet, goats fed less than 10 g kg diet exhibited pain in the extremities, swollen forefoot tarsal joints and skeletal deformations in the forelegs. These changes in bone suggest that vanadium may have a role that affects bone or connective tissue metabolism. This suggestion is supported by the finding that vanadium stimulated the mineralization of bones and teeth213 and the repair of bones.214 Orthovanadate stimulates bone cell proliferation and collagen synthesis in...

Debridement Of Neuropathic Ulcers

The commonest location in the front part of the foot, involving a metatarsal head, should be exposed after excision of nonvital tissues, with resection as far as the passage between the diaphysis and epiphysis with the oscillating saw and subsequent removal of the distal segment. The base of the phalanx is removed only if there is clinical or radiologic evidence of osteomyelitis. The bone segment removed is usually sent for microbiologic and histologic testing. Before suturing, we use high-pressure irrigation with physiologic saline and antiseptic we then perform careful hemostasis to avoid the risk of hematoma formation, and we introduce a suction drain or gauze which we remove after 24 hours. We suture the surgical access with a nylon or prolene 3-0 or 4-0 monofilament. We usually avoid suturing the subcutis with reab-sorbable sutures owing to the relative risk of ischemia and superinfection.

Growth Hormone Has Several Metabolic Effects

There are two principal mechanisms of bone growth. First, in response to growth hormone stimulation, the long bones grow in length at the epiphyseal cartilages, where the epiphyses at the ends of the bone are separated from the shaft. This growth first causes deposition of new cartilage, followed by its conversion into new bone, thus elongating the shaft and pushing the epiphyses farther and farther apart. At the same time, the epiphyseal cartilage itself is progressively used up, so that by late adolescence, no additional epi-physeal cartilage remains to provide for further long bone growth. At this time, bony fusion occurs between the shaft and the epiphysis at each end, so that no further lengthening of the long bone can occur.

Pharmacological Neuroanatomy

Serotonin-containing cells are concentrated in the pineal gland but are also found in discrete groups of cells in the midline regions of the pons and midbrain. The caudal cells project to the spinal cord and brain stem, while the rostral cell groups project to the limbic forebrain system, thalamus, neostriatum, and cerebellum. Raphe neurons receive dopaminergic input from the substantia nigra and the ventral tegmental area, norepinephrine input from the locus coeruleus, and other afferents from the hypothalamus, thalamus, and limbic forebrain. In the past decade, extensive pharmacological laboratory work has identified numerous serotonergic subsystems, including 5-HT1A-F , 5-HT2A-C , 5-HT3 , 5-HT4 , 5-HT5A-B , and 5-HT7 . More recently, specific receptor antagonists have also been developed that allow more refined pharmacological and neurochemical analysis of behaviors that relate to serotonin.

Indications in sleep disorders

Melatonin can promote more regular sleep-wake cycles in those who are blind and experience insomnia or excessive daytime sleepiness due to the failure to entrain their circadian rhythms to environmental light exposure. It can also be used to advance the sleep phase when it is taken in the evening in the delayed sleep-phase syndrome, and to retard sleep when it is taken in the morning in the advanced sleep-phase syndrome. Disorders of the pineal gland, such as pineal tumours, are usually associated with low endogenous melatonin secretion, and melatonin administration may be of benefit, acting as a hormone replacement or supplement treatment.

Endogenous disorders

Structural neurological conditions causing endogenous circadian rhythm disorders are uncommon. They may involve the suprachiasmatic nuclei or the pathways leading to and from these in the hypothalamus and from the pineal gland. The most important examples are as follows.

Brain injuries

Head trauma causes both primary brain injuries, such as haematomas and diffuse axonal injury, and secondary effects due, for instance, to hypotension, hypoxia, raised intracranial pressure and release of free radicals. Both fatigue and sleep disorders commonly follow closed head injuries. Obstructive sleep apnoeas are common in those who have had head injuries, but whether they are related in any way to the injuries is uncertain. Periodic limb movements have also been associated with head injuries, and a delayed sleep phase syndrome may develop, although more characteristically it follows neck injuries with disruption of the tract between the suprachiasmatic nuclei and the pineal gland. Deterioration in vision after head injuries may lead to a non-24-h sleep-wake rhythm, and drugs to treat for instance epilepsy or psychiatric disorders following the head injury may also cause sedation.

Normal Findings

Normally calcified structures are the pineal gland and the glomus of the choroid plexus in the trigone of the lateral ventricle. Calcification may be seen throughout the choroid plexus, including the temporal horns and the outlets of the fourth ventricle. Occasionally, the habenula (directly anterior to the pineal gland) may show calcification. The arteries of older patients are not uncommonly calcified. Calcification of the globus pallidus can be physiological and becomes more prominent with advancing age. Calcification in dural structures, including the walls of venous sinuses, is seen as a normal variant. Excessive calcifications of vascular or dural structures may indicate an underlying pathological process.


The injured child presents by refusing to use the affected limb but may not complain of pain. Often the shoulder is suspected to be the culprit. At presentation, the arm is held at the side with elbow partially flexed and the forearm pronated. Clinical findings include tenderness to palpation over the radial head and decreased range of motion at the elbow. Radiographs may show soft tissue swelling but are usually negative. Although the elbow is a commonly injured joint in children, interpretation of the radiograph may be difficult owing to joint anatomy. Because the radial epiphysis is not ossified, subluxation is diagnosed on clinical grounds.


Melatonin is a methoxyindole generated by enzymatic conversion of serotonin. It is synthesized and secreted principally by the pineal gland. Light is able to suppress or synchronize melatonin production. This explains the diurnal rhythm of melatonin secretion with a maximum during night time hours (02 00-04 00h) 320,321 . Melatonin receptors are present in different organs and involved in multiple physiologic functions 322 . The primary function of