Cure for Premature Ejaculation Discovered

Ejaculation Guru

In This Video You'll Discover: How I personally went from lasting less than 10 seconds in bed to over 30 minutes in bed. The real reason so many men suffer from premature ejaculation. And exactly what to do about it. How long you should be lasting if you want to truly satisfy a woman This, by the way, comes from a study carried out by a major University. The number #1 thing holding most men back from getting control over their orgasms and how you can change it. (By the way, most guys don't even realize this is holding them back, but it's critical to understand if you want to learn to last long in bed) What most porn stars will Never tell you about porn and its influence on your sexual stamina. The truth about penis size and its links with how long you last. What the number #1 reason is for relationships ending. and how premature ejaculation is critically linked to it. Why you should Avoid 99% of people trying to sell you long lasting condoms, creams or pills. Read more...

Ejaculation Guru Overview


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Ejaculation Trainer By Matt Gorden

Sick and tired of the humiliation of premature ejaculation? Drop everything and read every word on this page. the next few minutes could change your life completely. How You Can Last 10-30 Minutes Longer In Bed Tonight & Permanently End The Pain & Embarrassment Of Premature Ejaculation. You'll learn: Last longer in bed tonight, without creams, pills, or any other lame technique that doesn't work. Get a permanent improvement in your sexual stamina, regardless of how bad your premature ejaculation is now. Finally understand the root causes of Premature Ejaculation and cure yourself completely with a little knowledge and a few simple techniques. Read more...

Ejaculation Trainer By Matt Gorden Overview

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Premature Ejaculation

While ED is typically a dysfunction of older males, Premature Ejaculation (PE) most frequently occurs in younger men. PE is the persistent or recurrent ejaculation during sexual activity that is associated with minimal stimulation and individual or couple dissatisfaction with duration (ApA, 2000). At the present time, there are no objective criteria for determining the duration of sexual activity that constitutes premature ejaculation. Perhaps it is easier to describe what is not premature ejaculation Both partners agree that the quality of their sexual activities is not negatively impacted by efforts to postpone ejaculation (LoPic-colo, 1994). Prevalence estimates for PE derived from community samples indicate a rate of disorder between 36 and 38 (Spector & Carey, 1990). Definitive data on the etiology of premature ejaculation does not currently exist. Sociobiologists have theorized that it offers an evolutionary advantage and has been built into the human organism (Hong, 1984)....

Ejaculation and male orgasm

Most of the experimental work done so far for the investigation of ejaculation is based on behavioral experiments. Ejaculations in rats can be studied much the same way they are studied in humans, with the latency from first mount or intromission to ejaculation being the key variables (Fig. 1.3). Male rats typically ejaculate following several penile intromissions, and can ejaculate several times before becoming sexually exhausted, in which the male no longer responds to estrous odors or female solicitations. During successive ejaculatory series, the refractory period or post ejaculatory interval between each ejaculation and the subsequent resumption of copulation increases progressively. Penile intromission requires erection, and ejaculation typically requires sensory feedback from the penis that accumulates with multiple intromissions. The number of intromissions before ejaculation, the number of ejaculations achieved in a timed test, and the length of the post ejaculatory interval,...

Psychotherapy with rapid ejaculation

Cognitive behavioral therapy, as well as multimodal, psychodynamic and behavioral treatment is described in review papers however, there are no controlled outcome studies that examine the efficacy of these methods 13 . Masters and Johnson 3 utilizing multiple treatment techniques including the squeeze technique in combination with sensate focus and interpersonal therapy, reported failure rates of 2.2 immediately after treatment and 2.7 at the five-year follow-up. Other researchers have been unable to replicate Masters & Johnson's success rates. For instance, only 64 of men in Hawton's 14 study were characterized as successful in overcoming rapid ejaculation.

Pharmacologic treatment of premature ejaculation

Pharmacologic modulation of ejaculatory threshold represents a novel and refreshing approach to the treatment of PE and a radical departure from the psy-chosexual model of treatment, previously regarded as the cornerstone of treatment. The introduction of SSRIs has revolutionized the approach to, and treatment of, PE. Selective serotonin reuptake inhibitors encompass five compounds citalopram, fluoxetine, fluvoxamine, paroxetine and sertraline with a similar pharmacologic mechanism of action. Although the methodology of the initial drug treatment studies was rather poor, later doubleblind and placebo-controlled studies replicated the genuine effect of clomipramine and SSRIs to delay ejaculation. In spite of a development towards more evidence-based drug treatment research, the majority of studies still lack adequate design and methodology 44 . A recent meta-analysis of all drug treatment studies demonstrated that only 14.4 had been performed according to the established criteria of...

Male Orgasm

Male orgasm is a two-stage process involving emission (the movement of sperm into the urethra) and ejaculation (the explosive propulsion of the semen out of the urethra). How it is triggered is poorly understood although the inhibitory involvement of 5-HT is increasingly suggested by the pharmacology (described below). Neurons originating in the reticular paragigantocellularis nucleus of the ventral medulla (Fig. 6.1) and projecting to pudendal motor neurons and interneuronal areas of the lumbar cord appear to mediate inhibition of sexual reflexes and the majority of these fibers have been demonstrated to be serotonergic. Emission is a sympathetic response, integrated in the upper lumbar segments of the spinal cord it involves a sequence of contractions of smooth muscles of the epididymus, vas deferens, seminal vesicles and prostate to expel seminal fluid into the prostatic urethra. Various studies have demonstrated noradrenergic and cholinergic fibers in the epididymus, vas deferens...

Sexual function assessment in the male

The essential components of sexual function assessment in the male always include erectile response (onset, duration, progression, severity of the problem, nocturnal morning erections, self-stimulatory and visually erotic-induced erections), sexual desire, ejaculation, orgasm, sexually related genital pain disorders and partner sexual function, if available. Often, a dysfunction in one phase may precipitate a dysfunction in another. For instance, men with erectile dysfunction may report a loss of sexual desire or the onset of premature ejaculation.

Peter A Sargent and Guy M Goodwin Introduction

F52.4 Premature ejaculation F52.8 Other sexual dysfunction, not caused by organic disorder or disease F52.9 Unspecified sexual dysfunction, not caused by organic disorder or disease The pitfalls of medicalizing sexual dysfunction should be noticed here and have been illuminated in the lampoon by Szaz 2 he has interesting things to say in this area. Accepting the more conventional medical view, nevertheless, epidemiological studies suggest that sexual dysfunction is not uncommon in the general population. An analysis of 22 surveys of psychosexual dysfunction found inhibited sexual desire in 1-15 of men and in 1-35 of women, inhibited sexual excitement in 10-20 of men, premature ejaculation in 35 of men and inhibited orgasm in 5 of men and 5-30 of women.3 The scale and also the variability of these rates provides a potentially confounding background for the interpretation of findings of sexual dysfunction in specific disorders such as depression or as a consequence of treatment with...

Cell Divisions During Spermatogenesis

Cross Sectional Seminiferous Tubules

-Seminal vesicle -Ejaculatory duct deferens (about 10 per cent of the total), fluid from the seminal vesicles (almost 60 per cent), fluid from the prostate gland (about 30 per cent), and small amounts from the mucous glands, especially the bulbourethral glands. Thus, the bulk of the semen is seminal vesicle fluid, which is the last to be ejaculated and serves to wash the sperm through the ejaculatory duct and urethra.

Cellular Biology Of Prostatic Epithelium

The prostate of a 65-yr-old patient with benign prostatic hyperplasia (BPH) is shown. The prostate gland is composed of three anatomical zones. The central zone (C) located between ejaculatory ducts (D), and the proximal urethral segment (PU) accounts for about 25 of normal prostatic volume. Only about 10 of carcinomas arise in this zone. The transition zone (T) located around the proximal urethral segment accounts for about 5 of normal prostatic volume but steadily increases with age. Virtually all forms of BPH arise here. Transition zone cancer accounts for about 15-20 of prostatic adenocarcinoma and is commonly diagnosed in transurethral resection specimens from patients with BPH (incidental carcinoma). The peripheral zone (P) located around the distal urethral (DU) segment represents approx 70 of the normal gland. The majority (70-75 ) of prostatic adenocarcinomas and high-grade prostatic intraepithelial neoplasias (HGPINs) arise in this zone. CS,...

Rectification of terms

Noncontact erections (psychogenic erectile function responses to primary or secondary conditioned sexual cues) Copulatory measures latency to mount, intromit or ejaculate (shorter latency greater arousal) Enforced interval effect (model of premature ejaculation) Coolidge effect (increased arousal by changing sexual stimuli) Copulatory behavior after several ejaculatory series Estrus termination

Reproductive and Hormonal Functions of the Male and Function of the Pineal Gland

Two seminal vesicles, one located on each side of the prostate, empty into the pro-static end of the ampulla, and the contents from both the ampulla and the seminal vesicles pass into an ejaculatory duct leading through the body of the prostate gland and then emptying into the internal urethra. Prostatic ducts, too, empty from the prostate gland into the ejaculatory duct and from there into the prostatic urethra.

The squeeze technique

Concurrent with practicing the stop-start process, the couple is encouraged to spend time in mutual pleasuring involving non-genital massage and caressing, following the programme known as sensate focus. This is essential to reduce the genital-focused interaction of the ejaculatory control process. It is also essential for the man to be encouraged to satisfy the sexual needs of his partner during the training programme. Although success rates in the short term from 60 to almost 100 have been reported 11,69 , the methodology and design of these studies have been weak and fail to meet the criteria of evidence-based research. In addition, long-term maintenance of ejaculatory control induced by these treatment has shown to be very low 70 .

History and Epidemiology of Male Sexual Dysfunction

Proceeding into the beginning of the 21st century, new trends in sexual medicine promise to promote more popular interest and public discussion. A new and specific treatment for premature ejaculation, one of the most common forms of male sexual dysfunction, is in the final stages of clinical development and may be approved in 2007 19 . And an entirely new method of treating erectile dysfunction using the cutting-edge technique of gene therapy has just entered the initial stages of clinical development 20 . Importantly, progress in both male and female sexual health has been aided by the World Health Organization (W.H.O.) which has designated the years 2004 to 2009 for worldwide public emphasis on sexual health using the campaign slogan, Sexual Health a new focus for W.H.O The recognition by W.H.O. of the importance of sexual health is another signal of the vital role that sexual medicine is likely to play in the future.

Overlapping Diseases and Points of Confusion

Many men also confuse ejaculatory dysfunction (EjD), such as rapid or delayed ejaculation, with ED. Thus, the clinician must be keen and tease out the specific male sexual dysfunction. Indeed, EjD and ED may overlap, as depicted by Rosen et al. (Table 6.1) in the Multinational Study of the Aging Male 13 . This survey, which included postal questionnaires to men aged 50-80 in seven countries, demonstrated that EjD and ED were both highly prevalent and highly bothersome (Figs 6.1 and 6.2).

Type 5 Phosphodiesterase PDE5 inhibitors

Several authors have reported their experience with PDE5 inhibitors alone or in combination with SSRIs as a treatment for PE 62-66 . The proposed mechanisms for the effect of sildenafil on ejaculatory latencies include a central effect involving increased NO and reduced sympathetic tone, smooth muscle dilatation of the vas deferens and seminal vesicles, which may oppose sympathetic vasoconstriction and delay ejaculation, reduced performance anxiety due to better erections, and down-regulation of the erectile threshold to a lower level of arousal so that increased levels of arousal are required to achieve the ejaculation threshold. Most of these studies are uncontrolled and the results are confusing and difficult to interpret. The only double-blind placebo-controlled multicenter study showed no significant difference in the IELT of sildenafil-treated subjects compared to placebo, but did demonstrate significant improvements in the ejaculatory control domain and the ejaculatory function...

Models of male sexual dysfunctions

Premature ejaculation testing chamber by the experimenter after every intromission. In this way, Larsson was able to vary the time between intromissions and found that male intromission intervals that lasted longer than normal resulted in males that learned to ejaculate with far fewer intromissions. One of the interpretations of this data was that the imposition of longer intromission intervals made males more sympathetically aroused, and led to either a faster ejaculation or one that required less tactile penile stimulation. This model of hyperstimulation of sympathetic outflow by either highly stimulating, unpredictable, or stressful sex, formed part of the basis of Masters and Johnson's model of premature ejaculation a decade later. Despite this, the model has not been developed further, nor has it been used widely to examine drug effects. More recently it was reported that natural differences are found in the ejaculation latencies of male rats, which may indicate a more biological...

Ondemand treatment with selective serotonin reuptake inhibitors

Associated with less ejaculatory delay than daily treatment. Daily administration of an SSRI is associated with superior fold-increases in IELT compared to on-demand administration, due to greatly enhanced 5-HT neurotransmission resulting from several adaptive processes, which may include pre-synaptic 5-HT1A and 5-HT1B receptor desensitiza-tion 20 . On-demand treatment may be combined with either an initial trial of daily treatment or concomitant low-dose daily treatment 47-49 . A number of rapid acting short half-life SSRIs (Dapoxetine-Johnson & Johnson, UK-369,003-Pfizer) are under investigation as on-demand treatments for PE. Preliminary data suggest that dapoxetine (Johnson & Johnson) administered one to two hours prior to planned intercourse, is effective and well-tolerated, superior to placebo, and increases IELT two- to three-fold over baseline in a dose-dependent fashion 50 . In randomized, double-blind, placebo-controlled, multicenter, phase III, 12-week clinical trials...

The role of anxiety in sexual dysfunction

Men with premature rapid ejaculation try to distract themselves in an effort to prevent early ejaculation. They fear focusing on arousal, yet distraction does not effectively solve their problem. Counter intuitively, sex therapists teach men to focus on their arousal as a means of controlling ejaculation.

Ejaculation and Orgasm

Anorgasmia has been reported with all the commonly employed TCAs including clomipramine, imipramine, desipramine, nortriptyline and doxepine81,82 and MAOIs such as phenelzine.80 Rates of anorgasmia also vary widely in these studies. For example, in a study of sexual dysfunction in obsessive-compulsive disorder, 96 of patients treated with clomipramine described problems with orgasm 94 In another study 8.8 of patients treated with amitriptyline described delay in ejaculation.95 Clomipramine and SSRIs have been used with reported success to treat premature ejaculation (e.g., ref. 96). Other treatments for fluoxetine-induced anorgasmia that have been reported include yohimbine,78 and a number of direct and indirect dopaminergic agonists. These effects are probably a result of functional antagonism of the serotonergic action of fluoxetine (or other SSRIs). By blockade of a2-autoreceptors, yohimbine facilitates noradrenergic neurotransmission. The importance of dopaminergic input in...

Prevalence of hyperprolactinemia in other sexual dysfunctions

Routine determination of serum PRL in men consecutively seen for hypoactive sexual desire without ED (n 53), anorgasmia retarded ejaculation (n 74), and premature ejaculation (n 124) 210,249 found no HPRL in the two former sexual dysfunctions. However Schwartz etal. 250 reported on some male HPRLs revealed by isolated hypoac-tive sexual desire or anorgasmia. In contrast, serum PRL was mildly elevated (20-35ng mL) in 13 men with premature ejaculation (10 ). This was not the cause of sexual dysfunction since bromocriptine failed in every case to prolong the time to ejaculation. In addition serum T was normal in every case and no pituitary adenoma was detected in any patient.

Diagnosis of Hypoactive Sexual Desire

Some patients present themselves as having low sexual desire, which in fact is a result of another sexual dysfunction. Erectile dysfunction is sometimes confused by the patient as a sign of diminished desire. Likewise, the avoidance pattern that follows the frustration generated by a persistent dysfunction, like severe premature ejaculation or erectile dysfunction, can also be reported as absence of desire. These clinical situations demand a careful evaluation from the clinician before arriving at a clinical diagnosis.

Effects of Chemotherapy

PVB (cisplatin, vinblastine and bleomycin), which is used in patients with germ cell tumors, is a standard chemotherapy and results in minimal effects on long-term testicular function. Patients, however, can be affected by ejaculatory failure, caused by damage to the thoracolumbar sympathetic plexus during retroperitoneal lymph node dissection, and by preexisting germ cell defects. Hansen et al. found that whether patients were treated with orchiectomy, or with orchiectomy plus PVB, sperm production 1.5 years after treatment was similar. Approximately half in each group had sperm counts below the normal control reference level 28 . Lampe et al. analyzed the data on 170 patients with testicular germ cell cancers who had undergone treatment with either cisplatin or carboplatin-based chemotherapy 44 .After median of 30 months from the completion of chemotherapy, they discovered that azoospermia occurred in 54 (32 ) of the patients and oligospermia occurred in 43 (25 ). The probability of...

Diagnosis and Evaluation

The diagnostic evaluation of ejaculatory dysfunction focuses on finding potential physical and specific psychologic learned causes of the disorder. A medical (particularly genito-urinary) examination and history are critical, as these may uncover physical anomalies, various pathophysiologies, and iatrogenic procedures associated with the IE. In addition, concomitant or contributory neurologic, endo-crinergic, or erectile disorders can be identified and addressed. Particular attention should be given to identifying reversible urethral, prostatic, epididy-mal, and testicular infections. Given the lack of understanding of basic physiologic mechanisms responsible for the timing of ejaculation, assessment procedures tapping into the basic physiology of the ejaculatory reflex (e.g. sensory thresholds or efferent reactivity) have to date not been particularly meaningful or useful for the management of this dysfunction.

Actions of Drugs at 5HT1 Receptors

Systemic administration of 5-HT1A receptor agonists such as 8-OH-DPAT in rats reduces penile erections induced by 5-HT2C agonists36 and facilitates ejaculation by decreasing ejaculatory threshold and latency (Table 6.1).37 Administration of 8-OH-DPAT causes a biphasic dose-response pattern in the rhesus monkey. Low doses facilitated ejaculation by reducing ejaculatory threshold and latency, perhaps by a preferential presynaptic action as seen in other models of 5-HT1A receptor function,38 and high doses interfered with copulation and ejaculation.39

Germ Layer Lineage Stem Cells

Because of its developmental lineage (see Fig. 1), the germ layer lineage mesodermal stem cell has the potential to form cells of the adrenal cortex, Sertoli cells, interstitial cells of Leydig, ovarian stroma, follicular cells granulosa cells, thecal cells, skeletal muscle, smooth muscle, cardiac muscle, unilocular adipocytes, multilocular adipocytes, fibrous connective tissues, dermis, tendons, ligaments, dura mater, arachnoid mater, pia mater, organ capsules, organ stroma, tunica adventitia, tunica serosa, fibrous scar tissue, hyaline cartilage, articular cartilage, elastic cartilage, growth plate cartilage, fibrocartilage, endochondral bone, intramembranous bone, arterial endothelial cells, venous endothelial cells, capillary endothelial cells, lymphoidal endothelial cells, sinusoidal endothelial cells, erythrocytes, monocytes, macrophages, T-lymphocytes, B-lymphocytes, plasma cells, eosinophils, basophils, Langerhans cells, dendritic cells, natural killer cells, bone marrow...

Prevention of Testicular Damage

Ejaculatory azoospermia is not the same as testicular azoospermia 14 . Hence,studies on the gonado- toxicity of chemotherapy have to be interpreted in light of the fact that assisted reproductive technology makes it possible to use testis sperm to conceive. The level of sperm necessary for sperm to exist in the testis is far less than the level required for sperm in the ejaculate 14 . As a result, testis sperm extraction (TESE), followed by ICSI, now makes it possible for patients who have azoospermia on semen analysis, and did not sperm bank, to father children. A retrospective study by Damani et al. evaluated 23 men with ejaculatory azoospermia and a history of chemotherapy. All men underwent TESE in search of usable sperm. Spermatozoa were found in 15 (65 ). The subsequent fertility rate was 65 and pregnancy occurred in 31 of cycles 21 . This illustrates the importance of performing a full evaluation on men with post-chemotherapy azoospermia before diagnosing them as sterile.

The Use of SSRIs to Treat Conditions Other Than Depression

One of the major spin-offs to occur as a result of the overwhelming commercial success of the SSRIs has been the spread of their use not only for severe depression to mild dysthymia, but also to other psychiatric, e.g., bulimia, panic disorder, obsessive-compulsive disorder, anxiety, and non-psychiatric conditions, e.g., obesity, premature ejaculation, Raynaud's syndrome, headache. Finally, the pharmaceutical industry, ever adept at turning a disadvantage into an asset, have responded to the increased reporting of SSRI-induced male sexual dysfunction by putting these drugs into clinical development for the treatment of premature ejaculation.

Male infertility

Approximately 98 per cent of males with CF are infertile. The genetic abnormality that results in CF is associated with aberrant embryological development of the reproductive portion of the mesonephric (wolffian) duct. At birth, this results in variable absence of the vas deferens, seminal vesicle, ejaculatory duct and body and tail of the epididymis3 (see Figure 11.2). While active spermatogenesis occurs in the testis, sperm are unable to be transported from the testis due to congenital absence of the vas deferens (Kaplan etal. 1968 McCallum etal. 2000). Neither sex hormone production nor sexual function are affected.

Future aspects

Except where a treatable organic cause has been identified or a drug known to impair ejaculatory function has been withdrawn, the mainstay of treatment is by psychotherapy, but this is not evidence-based, as the most successful psychotherapeutic intervention has not been established. Future research should address this shortcoming. A number of pharmacologic agents are reported to treat drug-induced delayed (inhibited) ejaculation, but these observations are not based on well-controlled and adequately powered studies, but rather on single cases or small series of cases. The pharma-cotherapy of inhibited ejaculation requires proper evaluation.

Painful ejaculation

Characterized syndrome and may be associated with benign prostatic hypertrophy (BPH), infection, or inflammation from acute prostatitis, chronic pro-statitis chronic pelvic pain syndrome, seminal vesi-culitis, seminal vesicular calculi, or ejaculatory duct obstruction, a treatable cause of male infertility 122-125 . Nickel reported that 18.6 of men with lower urinary tract symptoms (LUTS) diagnosed with clinical benign prostatic hyperplasia reported painful ejaculation 123 . Men with BPH and painful ejaculation have more severe LUTS and reported greater bother, and had a higher prevalence of erectile dysfunction and reduced ejaculation, than men with LUTS only 78 . Treatment of men with LUTS with a-blocking drugs may be associated with painful ejaculation. A lower incidence of pain has been reported with the uroselective a-1 blocking drug, alfuzosin 126 .


More difficult to identify are inherent physiologic factors that account for variation in ejaculatory latency and thus might play a role in IE (particularly primary anorgasmia). Low penile sensitivity, most often associated with aging, may exacerbate difficulty with reaching orgasm, but it is unlikely to be a primary cause 86,87 . Alternatively, variability in the sensitivity of the ejaculatory reflex may be a factor, as several studies have demonstrated shorter latencies and stronger bulbocavernous EMG and ERP responses in men with ejaculation. However, ejacu-latory response and latency are more likely to result from an interaction between higher cortical (cognitive-affective-arousal), supraspinal, spinal, and peripheral neuron systems 88 .


For example, has emphasized the need to remove the demand (and thus anxiety-producing) characteristics of the situation, noting that men with IE may be over-conscientious about pleasing their partner 89 . This ejaculatory performance anxiety interferes with the erotic sensations of genital stimulation, resulting in levels of sexual excitement and arousal that are insufficient for climax (although more than adequate to maintain their erections).

Thyroid hormones

330 found ED in 64 and 15 , respectively (associated with hypoactive sexual desire and retarded ejaculation in most of the hypothyroid patients), and premature ejaculation in 7 and 50 , respectively. After return to normal levels of serum thyroid hormones for eight to 12 weeks, the prevalence of premature ejaculation fell from 50 to 15 in the hyperthyroid patients, while that of retarded ejaculation was reduced by half in those with hypothy-roidism. The mean ejaculation latency time doubled in the former and significantly decreased in the latter, suggesting the possibility of a direct involvement of thyroid hormones in the physiology of ejaculation. In 38 patients referred for ED and found to be hypothyroid, Baskin 331 reported that erectile function returned in the majority following thyroid hormone substitution. Conversely Wortsman et al. 332 did not observe any sexual improvement following thy-roxin treatment in a short series of ED patients with primary hypothyroidism. In...


4 Patient role-playing an exaggerated ejaculatory response on his own and in front of his partner. This can be especially helpful where the man's inability to ejaculate arises from the embarrassment the patient thinks he will experience when he loses emotional control during ejaculation.


However, in a research setting, objective measurement of IELT by stopwatch and subjectively validated, reliable and consistent patient-reported outcome measures (PROs) of ejaculatory control, sexual satisfaction, and bother distress, are essential in studies assessing treatment 26-28 . Voluntary control Various authors have proposed the extent of voluntary control over ejaculation as an appropriate measure, suggesting minimal or absent control as defining PE 34-36 . However, the patient's feeling of ejaculatory control is a subjective measure and difficult to translate in quantifiable terms. Although feelings of ejaculatory control are part of the ejaculation process, diminished feelings of ejaculatory control are not exclusive for men suffering from PE. Consistent with this, attempts to operationalize the dimension of control has reported conflicting results, making comparison across subjects or across studies difficult 37-39 . Nevertheless the dimension of voluntary control over...


It is likely that only some men seeking treatment for PE require in-depth psychotherapy. In spite of hard evidence on the efficacy of psychotherapy, behavioral retraining is still often practiced by sexologists. Behavioral treatment is distinguished in the stop-start and the squeeze 11,68 . The basis of behavioral retraining is the hypothesis that PE occurs because the man fails to appreciate the sensations of heightened arousal and recognize the feelings of ejaculatory inevitability.


The patient (and partner) histories are the key toward the successful diagnosis of male ED. ED is highly prevalent in men with a variety of medical diseases, including depression, and is often confused with ejaculatory disorders. The clinician should have a high index of suspicion when evaluating men with these medical diseases for ED. The clinician must also be aware of the psychologic aspects of male ED, with a referral to a sex or marital therapist indicated as necessary. Finally, there is an evolving paradigm that ED may be a predictor

Closing Comments

Although recent neuropharmacologic studies and animal research has deepened our understanding of neurobiology of PE and the mechanism of pharma-cotherapy, a genuine understanding of the etiology of lifelong and acquired premature ejaculation is still lacking. Despite high levels of evidence from multiple well-controlled studies to support the efficacy of drug treatment strategies in lifelong PE, there is little or no evidence to support the role and longitudinal efficacy of behavioral therapy. As such, the place of


Premature ejaculation is claimed to be the most common male sexual disorder, affecting 5-40 of sexually-active men 9 . It is believed that there is a higher frequency of PE in adolescents or young adults. Premature ejaculation is more frequently reported by men in East Asia (China, Indonesia, Japan, The current knowledge of the epidemiology of PE is limited by both the lack of a consensus definition of lifelong PE and a failure to consistently distinguish lifelong and acquired PE in most earlier studies. Medical literature contains several univariate and multivariate operational definitions of premature ejaculation. The lack of agreement as to what constitutes premature ejaculation has hampered basic and clinical research into the etiology and management of this condition. Premature ejaculation has been defined in various ways. Masters and Johnson defined PE as . . . the inability to delay ejaculation long enough for the woman to achieve orgasm fifty per cent of the time 11 . On the...


In males and females, orgasm is characterized by a peak in sexual pleasure that is accompanied by rhythmic contractions of the genital and reproductive organs, cardiovascular and respiratory changes, and a release of sexual tension. In males, orgasm generally occurs in two stages emission, which refers to rhythmic muscular contractions that force semen into the ejaculatory ducts, and expulsion, which is the release of semen through the urethra (ejaculation). Unlike males, some females (approximately 15 ) are able to experience multiple orgasms, and some women experience orgasm and perhaps ejaculation when the Grafenberg spot, an area along the anterior wall of the vagina, is stimulated. Contrary to Freud's assertion of two distinct types of orgasm in females, clitoral (the infantile orgasm) and vaginal (the mature orgasm), Masters and Johnson (1966) found no physiological differences in orgasm produced by vaginal versus clitoral stimulation. Other researchers note that intensity of...

5 Secrets to Lasting Longer In The Bedroom

5 Secrets to Lasting Longer In The Bedroom

How to increase your staying power to extend your pleasure-and hers. There are many techniques, exercises and even devices, aids, and drugs to help you last longer in the bedroom. However, in most cases, the main reason most guys don't last long is due to what's going on in their minds, not their bodies.

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