Cure for Chronic Fatigue Found
A number of people are diagnosed with the general label chronic fatigue syndrome. Seventy percent of people who suffer from chronic fatigue are women under age forty-five. Many of them may be misdiagnosed with chronic fatigue when, in fact, they are hypothyroid. In general, chronic fatigue is still a bit of a mystery, and much of the current research points to sleep deprivation, often with unrecognized sleep disorders as the main cause. This section briefly summarizes the accepted theories and definitions. Chronic fatigue syndrome (CFS) has been around longer than you might think. In 1843, for example, a curious condition called fibrositis was described by doctors. It was characterized by symptoms similar to those now seen in fibromyalgia (chronic muscle and joint aches and pains) and CFS. The term rheumatism, now outdated, was frequently used as well to describe various aches and pains with no specific or identifiable origin. In the early 1980s, two physicians in Nevada who treated a...
Although diseases similar to chronic fatigue syndrome (CFS) have been recognized for centuries under various rubrics, strict diagnostic criteria for conditions dominated by medically unexplained chronic fatigue were first proposed in 1988. Current diagnostic criteria were crafted in 1994 by an International Chronic Fatigue Syndrome Study Group as an attempt to standardize patient populations included in research studies. The International CFS Re
Sleep is usually felt to be light and unrefreshing and this correlates with the number of tender points. Tiredness is common during the day, but it is unusual for sleep to be entered. Fibromyalgia may have a similar pathogenesis to the chronic fatigue syndrome, with hyperarousal, changes in endocrine function and in autonomic responses, abnormal reactions to stress, and a particularly increased sensitivity to pain.
One impetus for the idea of allostasis was linked to concern about our social world. In a paper by Eyer and Sterling (1977) entitled Stress-Related Mortality and Social Organization, a major portion was a critique of our society and the onset of a variety of disease states. Sterling and Eyer (1988) and others pointed to the detrimental sequelae of chronic arousal (Chrousos and Gold, 1992, 1998 McEwen and Stellar, 1993 Schulkin et al., 1994 Goldstein, 1995a, b). Sterling and Eyer were concerned about widespread chronic fatigue due to overstimulation. They endorsed practices that enhance calmness, such as transcendental meditation and community-based attachments.
Rehearsal Enhances the Transference of Short-Term Memory into Long-Term Memory. Psychological studies have shown that rehearsal of the same information again and again in the mind accelerates and potentiates the degree of transfer of short-term memory into long-term memory and therefore accelerates and enhances consolidation. The brain has a natural tendency to rehearse newfound information, especially newfound information that catches the mind's attention. Therefore, over a period of time, the important features of sensory experiences become progressively more and more fixed in the memory stores. This explains why a person can remember small amounts of information studied in depth far better than large amounts of information studied only superficially. It also explains why a person who is wide awake can consolidate memories far better than a person who is in a state of mental fatigue.
Growth hormone (GH somatotropin) is secreted by the anterior pituitary in response to hypothalamic GH-releasing hormone (GHRH). GH promotes linear growth and has both anabolic and catabolic effects. Hypersecretion of GH causes gigantism when it occurs before epiphyseal closure and acromegaly when it begins afterward. Hyposecretion causes short stature in childhood and possibly a chronic fatigue-like syndrome in adults. GH acts indirectly through insulin-like growth factors (IGFs, somatomedins), with IGF-1 (formerly somatomedin C) being the most important for growth. Prolonged exposure to elevated levels of GH and IGF-1 results in the insidious onset of skeletal and soft tissue overgrowth, the latter of which is most pronounced in tissues containing large amounts of cartilage proteoglycans. '109 Cortical bone density is increased and trabecular bone (e.g., vertebral) density is decreased, probably because of coexistent hypogonadism. '109 Soft tissue hypertrophy may result in...
The number of R. slovaca infections in Europe is still, probably, under-evaluated. From January 1996 to April 2000, its role in this syndrome was evaluated in 67 patients from France and Hungary presenting with TIBOLA (112). A total of 17 cases of R. slovaca infection were confirmed in this cohort by molecular methods. Recently, 14 patients were reported from southern France. It has been suggested that R. slovaca may be responsible for as much as 25 of SFG rick-ettsioses in this region (113). Infections were most likely to occur in children and in patients bitten by a tick during the colder months of the year. Fever and rash were uncommon and sequelae included localized alopecia at the bite site and chronic fatigue. Similar cases have also recently been reported in Bulgaria (114) and Spain (115). Finally, the isolation of R. slovaca from a patient has been recently reported, providing definitive evidence that R. slovaca is a human pathogen (116).
In Wilson's syndrome literature such as his book Wilson's Thyroid Syndrome A Reversible Thyroid Problem (1991), published by the Wilson's Syndrome Foundation (which also appears on the Wilson's Syndrome website), more than thirty-seven symptoms as well as others are listed as part of the condition. There are few people, healthy or not, who could say that they don't have at least a few of the symptoms on the list. And while some of these symptoms can be due to genuine hypothyroidism, some of the symptoms describe completely unrelated problems, such as asthma. Wilson's list of symptoms also mimic those that you'll find under the following conditions neurasthenia, chronic fatigue, fibromyalgia, multiple chemical sensitivity, chronic Epstein-Barr disease, and chronic candidiasis. Since these conditions typically describe people with chronic malaise, in search of an answer to their malaise, it is suspicious and convenient that Wilson's syndrome seems to offer an answer which, however, is...
Should also be distinguished from mental fatigue with poor concentration or motivation, physical weariness or fatigue which usually has an organic cause or may be related to insomnia, and the feeling of subalertness which may be due to a reduced wakefulness drive or an increased drive to sleep, as in narcolepsy. The duration, frequency and timing of naps should be noted, and whether or not these are refreshing. Many patients with narcolepsy feel refreshed after a nap of 5-30 min, whereas in idiopathic hypersomnia naps are longer but are unrefreshing.
In recent years, it became in vogue for thyroid patients to request supplementation with a combination of T3 (Cytomel) and T4 (levothyroxine sodium). Certain books and articles about T3 supplementation reported that it helped with brain fog, depression, and other apparent symptoms of hypothyroidism. There was also a trend that began in psychiatry, where T3 was added to antidepressants, which reportedly helped alleviate depression, but which has been disputed by most endocrinologists as not helpful and potentially harmful.
Sleep paralysis should be distinguished from the generalized fatigue on waking which is common in the chronic fatigue syndrome and depression and is due to a lack of motivation rather than true paralysis. This lasts longer than sleep paralysis and small movements, for instance of the fingers, are possible. Sleep paralysis may also be confused with atonic seizures, transient cerebrovascular attacks and psychogenic disorders.
Physical fatigue related to muscle weakness is present in around 75 of those with multiple sclerosis but this should be distinguished from mental fatigue and from EDS. The latter is usually due to sleep fragmentation caused by immobility, discomfort, muscle spasms, nocturia and occasionally central or obstructive sleep apnoeas. Periodic limb movements in sleep have also been reported as being common in multiple sclerosis, and depression may have an important impact on sleep. Polysomnography may show an increased sleep latency and duration of time awake after sleep onset, and early morning awakening. The HLA DR2 type is present in 50-60 of those with multiple sclerosis, but narcolepsy, which shares this HLA type, is not commoner in multiple sclerosis than in the general population.
The evolution of acute Q fever to chronic fatigue syndrome has been described in Australia (15) and in the United Kingdom (16). Ayres et al. (15) interviewed 71 patients five years after the acute phase, and compared them with controls. The patients reported more frequently sweat, dyspnea, blurred vision, and abnormal tiredness. Penttila et al. have demonstrated that the patients with a chronic fatigue syndrome (20 ) show moderate abnormalities of their cytokines regulation (16). A geographical variation seems to exist, since none of the 80 patients followed-up in Martigues (southern France) after an acute Q fever developed chronique fatigue syndrome, versus 37 in an English study (17).
The hypothalamic-pituitary-adrenal axis (HPAA) has been of particular interest as the major pathway through which the brain responds to psychological stress. Corticotropin-releasing factor (CRF) from cells of the par-aventricular hypothalamus acts as a releasing hormone for adrenocorticotropin (ACTH) in the pituitary, and ACTH releases corticosteroids from the adrenal cortex. Cortico-steroids (cortisol, corticosterone) are essential for the body and allow psychological and physical adaptation to stressors. In addition, they exert a negative feedback on the release of CRF and ACTH. Disturbances of the HPAA are associated with both mental and physical illness. CRF can easily be activated by stress and promotes depression, anxiety, and other forms of behavioral pathology. By activating the autonomic nervous system, CRF further links psychological stress to gastrointestinal and cardiovascular disorders. Although homeostasis of the HPAA is maintained by negative feedback, permanently...
Excessive daytime sleepiness is common when a large volume of brain is irradiated, for instance with prophylactic treatment for leukaemia or small cell carcinomas of the bronchus, whereas localized radiotherapy even close to the hypothalamus for pituitary tumours causes less EDS. Excessive daytime sleepiness following cranial irradiation should be distinguished from the physical fatigue that follows radiotherapy to areas outside the brain.
Clinical Features and Associated Findings. Infectious mononucleosis is the only known disease caused by acute EBV infection. The neurological complications of infectious mononucleosis include meningoencephalitis, Guillain-Barre syndrome, cerebellitis, transverse myelitis, cranial nerve palsies of which the most common is a seventh nerve palsy, and a variety of optic nerve abnormalities (papilledema, optic neuritis, retrobulbar neuritis). EBV infection is almost universally associated with primary central nervous system lymphomas in patients with AIDS.'wi Persistent viral activity or reactivation of EBV has been studied in the etiology of chronic fatigue syndrome. Chronic Fatigue Syndrome. Chronic fatigue syndrome is characterized by debilitating fatigue accompanied by a variety of symptoms including cognitive difficulty, The diagnosis of primary CNS lymphoma is based on CT or MRI evidence of a focal enhancing mass lesion or lesions, and typically an unsuccessful response to antitoxo-...
If you look at the list of symptoms in Chapter 3 that comprise hypothyroidism, as well as the symptoms of exhaustion in thyrotoxicosis (see Chapter 4), many of them can exacerbate normal fatigue that is part of daily life. Hypothyroid symptoms can also be confused with chronic fatigue. This chapter discusses normal versus unusual or chronic fatigue, and suggests ways to manage both fatigue that is thyroid-related and fatigue that is unrelated to but can aggravate your thyroid condition.
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